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The peptide does not suddenly become useless on day 29

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a and b : Principal component analysis of liver ( a ) and skeletal muscle gene expression ( b )

BPC-157s primary signalling mechanisms are well-characterised: it activates VEGFR2, drives downstream ERK1/2 phosphorylation, and stimulates nitric oxide synthesis via the Akt-eNOS axis producing pro-angiogenic, fibroblast-activating, and extracellular matrix-stabilising effects in experimental models