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These are research chemicals

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found that TMAO induced mitochondrial dysfunction, thereby decreasing contractility of cardiomyocytes (100)

Its receptor, GLP-1R is a member of class B GPCRs (G protein-coupled receptors) which are characterized by a seven transmembrane domain at their C-termini and a large extra-cellular domain at their N-termini 27,28,29,30

While the drugs are different, their similar mechanisms make them eligible to be consolidated into an MDL