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Its full name, "Body Protection Compound," hints at its origins as a protective substance in the digestive system

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This finding could be explained by a competition among selenoproteins for available selenium in the form of selenocysteine required for biosynthesis of the selenoproteins and for the selenium-incorporation machinery

These findings suggest that TMAOs effects on metabolic function are contingent on specific contexts

Thats short enough to make sustained IGF-1 receptor activation difficult to study in vivo